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Arguments against FPHL representing the female counterpart of male AGA are a probably mother-to-daughter transmission of FPHL, a significantly lower incidence of FPHL in women than AGA in men (), occurrence of FPHL in the absence of circulating androgens (), lack of response to antiandrogen therapy in normoandrogenemic premenopausal women (), lack of response to 1 mg oral finasteride daily in postmenopausal women (), and occurrence of male pattern AGA in women with pathologically elevated androgen levels.

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